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Abd El-Mageed et al
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The common interactions involve two salt bridges with glutamic acid residues at positions 6.53b and 7.42b via the positively charged N-terminal nitrogen atom of tyrosine 1, stacking interactions with tryptophan residues via phenylalanine 22, and multiple hydrogen bonds with various conserved residues across all three receptor subtypes [3]
By simultaneously influencing GLP-1 and GIP receptors, it not only curbs appetite but also improves metabolic functions
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